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Papers on High Blood Pressure

Relationship Between Blood Pressure and Stroke Risk in Patients With Symptomatic Carotid Occlusive Disease
P.M. Rothwell, S.C. Howard and J.D. Spence
Stroke 2003;34;2583-2590; originally published online Oct 30, 2003;
DOI: 10.1161/01.STR.0000094424.38761.56
Stroke is published by the American Heart Association. 7272 Greenville Avenue, Dallas, TX 72514
Copyright © 2003 American Heart Association. All rights reserved. Print ISSN: 0039-2499. Online
ISSN: 1524-4628


ImageBackground and Purpose—Blood pressure lowering in patients with a previous transient ischemic attack (TIA) or stroke reduces the risk of recurrent stroke and coronary vascular events. However, there is uncertainty about the risks and benefits in patients with severe carotid occlusive disease, particularly those with a carotid occlusion or bilateral > 70% carotid stenosis in whom cerebral perfusion is often impaired and may depend directly on systemic blood pressure. Therefore, we studied the effect of carotid artery disease on the relationship between blood pressure and stroke risk in patients with recent TIA or stroke.

 

Blood pressure measuring devices: recommendations of the European Society of Hypertension
Eoin O'Brien, Bernard Waeber, Gianfranco Parati, Jan Staessen, Martin G Myers
BMJ 2001;322;531-536
doi:10.1136/bmj.322.7285.531

ImageThere is a large market for blood pressure measuring devices not only in clinical medicine but also among the public where the demand for self measurement of blood pressure is growing rapidly. For consumers, whether medical or lay, accuracy should be of prime importance when selecting a device to measure blood pressure. However, most devices have not been evaluated for accuracy independently using the two most widely used protocols: the British Hypertension Society (BHS) protocol and the standard set by the US Association for the Advancement of Medical Instru­ mentation (AAMI).1 2 The Working Group on Blood Pressure Monitoring of the European Society of Hypertension has decided to review blood pressure measuring devices regularly to guide purchasers.3 For this first report devices for which there is published evidence of independent validation using these proto­ cols have been surveyed. Because most blood pressure devices have not been independently validated, only a fraction of the many devices available have been surveyed. Devices that have been validated recently for which results have not yet been published were not included, but this shortcoming should be addressed in future.

 

Association of Pulse Pressure With Cardiovascular Outcome Is Independent of Left Ventricular Hypertrophy and Systolic Dysfunction: The Strong Heart Study
Vittorio Palmieri, Richard B. Devereux, Jacqueline Hollywood, Jonathan N. Bella, Jennifer E. Liu, Elisa T. Lee, Lyle G. Best, Barbara V. Howard, and Mary J. Roman

Background: Whether increased pulse pressure (PP) predicts cardiovascular (CV) events independent of left ventricular hypertrophy (LVH) and systolic dysfunction is unclear.

 

Assessment of blood pressure control in hypertensive stroke survivors: an ambulatory blood pressure monitoring study
Nikolaos Zakopoulos, Konstantinos Spengos, Georgios Tsivgoulis, Vassilios Zis, Efstathios Maniosa and Konstantinos Vemmos

Background We compared the sensitivity of office blood pressure and ambulatory blood pressure monitoring recordings in evaluating the effectiveness of antihypertensive treatment and identified factors related to inadequate blood pressure control among hypertensive stroke survivors.

 

Arterial Stiffness and Hypertension
Donna K. Arnett, PhD
Division of Epidemiology, School of Public Health, University of Minnesota, USA


OVERVIEW OF THE ROLE OF ARTERIAL STIFFNESS IN HYPERTENSION
Historically, arteries were considered to be passive conduits of blood; today, they are viewed as complex, active participants in cardiovascular function, including abnormalities in blood pressure. Stiffening of large arteries may be both a cause and a consequence of hypertension. There are several studies, including studies done by the University of Minnesota, that confirm that as arterial pressure rises, acute and reversible stiffening of the large arteries occurs without a change in the structure of the artery. Arterial stiffness increases transiently as blood pressure rises. Arterial stiffening also increases because of the structure of the artery changes. Persistently elevated blood pressure accelerates atherosclerosis, arterial smooth muscle hyperplasia and hypertrophy, and collagen synthesis, thereby increasing arterial stiffness.

 
 
 

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